Semax / Selank Blend
Dual Neuropeptide Research Blend
$100.00
- FormatLyophilized powder
- StorageStore lyophilized vial frozen; refer to COA for lot-specific stability data
100 in stock
About Semax / Selank Blend
The Semax / Selank Blend combines two seven-amino-acid peptides developed through research originating from the same Russian institute. Semax includes a fragment derived from ACTH along with a short stabilizing sequence that helps protect it from rapid enzymatic breakdown, and has been studied for its effects on BDNF and dopamine and serotonin signaling. Selank is derived from tuftsin, a naturally occurring immunomodulatory peptide, shares the same stabilizing C-terminal sequence, and has been investigated for its effects on GABA-related signaling and stress-associated behavioral and physiological endpoints.
Full Specification
| Molecular Formula | Semax: C37H51N9O10S · Selank: C33H57N11O8 |
| Molecular Weight | Semax: 813.93 g/mol · Selank: 751.88 g/mol |
| Purity | ≥99% HPLC |
| Appearance | White to off-white lyophilized powder |
| Storage | Store lyophilized vial frozen; refer to COA for lot-specific stability data |
| Format | Lyophilized powder |
The Semax / Selank Blend Molecule
An illustrative representation generated from the compound's molecular formula, alongside its logged molecular data. This figure is schematic — not a measured conformation.
How Semax / Selank Blend Works
The pathways Semax / Selank Blend acts on — and what each one does. The animation traces its signal outward from the compound to every target it engages.
Signalling pathways studied in preclinical models · illustrative
Shared Structural Lineage
Common C-Terminal SequenceSemax and Selank share the same stabilizing C-terminal sequence, which helps resist enzymatic breakdown, and originate from the same Russian research program despite acting through different primary pathways.
Semax: ACTH-Derived Neurotrophic Signaling
NeurotrophicSemax includes an ACTH-derived fragment and has been studied for effects on BDNF along with dopamine and serotonin signaling in rodent models, without showing the stress-hormone activity of ACTH in experimental studies.
Selank: GABA-A Receptor Modulation
GABAergicSelank has been studied for its ability to influence GABAergic signaling, including GABA-A receptor activity and receptor-subunit expression, pathways involved in inhibitory neurotransmission and stress-related responses.
Selank: BDNF & Enkephalin Regulation
NeurotrophicSelank has been associated with changes in BDNF expression and enkephalin metabolism, systems that contribute to neuroplasticity, synaptic function, and mood- and cognition-related signaling.
Selank: Tuftsin-Derived Immune Modulation
ImmunomodulatoryDerived from the natural immune peptide tuftsin, Selank has been investigated for its effects on cytokine and interferon signaling, pathways involved in coordinating inflammatory and immune responses.
What Semax / Selank Blend Is Researched For
Neuronal Growth Research
Semax has been studied for effects on BDNF, associated with neuronal growth and plasticity.
Neurotransmitter Signaling
Semax has been examined for effects on dopamine and serotonin signaling in rodent models.
Anxiety-Related Signaling
Selank has been studied in preclinical anxiety models, including elevated plus maze and open-field testing, with effects linked primarily to GABAergic neurotransmission.
Learning & Memory
Selank has been examined in animal models of stress-related cognitive impairment, focusing on learning and memory consolidation.
Cytokine & Immune Regulation
Selank has been investigated for its influence on cytokine and interferon signaling, including pathways involving IL-6 and TNF-α.
Transcriptomic Effects
Selank-associated gene-expression changes have been identified in pathways related to serotonin and dopamine metabolism, synaptic function, and neuroplasticity.
Common Questions About Semax / Selank Blend
A combination of Semax and Selank, two seven-amino-acid neuropeptides from the same research lineage, studied through complementary signaling pathways.
A seven-amino-acid peptide including an ACTH-derived fragment, originally developed by Russian researchers and studied for BDNF and dopamine/serotonin signaling.
A synthetic seven-amino-acid peptide derived from tuftsin, studied for GABA-related signaling and stress-associated endpoints.
They share a stabilizing structural sequence but act on different primary pathways (BDNF/neurotransmitter signaling vs. GABAergic modulation), making the pair useful for combined-pathway research.
No, despite being derived in part from ACTH, it has not shown the same stress-hormone activity in experimental studies.